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Other Oxford events

Healing Hearts: Advancing cardiomyopathy research, one beat at a time

Please note this event takes place on the first floor and has no step-free access.
Past event - 2024
15 May Doors 7pm
Event 7.30pm to 9.30pm
St Aldates Tavern, 108 St Aldate's,
Oxford OX1 1BU
Sold Out!
Join us for an enlightening session sponsored by CureHeart, dedicated to exploring cutting-edge research in the field of cardiomyopathy. Our distinguished speakers, Professor Hugh Watkins, Dr Rosie Kirk, and Dr Miguel Varela, will share their expertise and insights into the latest advancements aimed at curing inherited heart muscle diseases.

 

Genes, Families & Fixing Broken Hearts

Hugh Watkins (British Heart Foundation Professor of Cardiovascular Medicine)
Inherited heart muscle diseases or cardiomyopathies affect 1 in 250 people worldwide and run in families. Often the first time a family learns that they have this condition is when a family member dies unexpectedly. Hear how we aim to cure this devastating condition using cutting edge gene editing technology.
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My Heart Will Go On: Solving a Titin-ic problem

Rosie Kirk (DPhil Student)
Titin is the largest protein in the body, and it serves a crucial purpose providing structure to our heart cells. Genetic mutations that run in families can reduce Titin levels in the heart and cause dilated cardiomyopathy, a common and potentially devastating disease of the heart muscle that currently has no cure. In this talk you will learn about the obstacles to creating gene therapies targeting this giant protein, and current research being undertaken to get closer to a cure.
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A Disease of the Heart that Cannot Be Cured Only with Gold

Miguel Varela (Lead Research Scientist)
We are up to the challenge of penetrating plasma membranes, crossing the endothelium of blood vessels, and resisting extracellular nuclease degradation until we reach your heart. Let's escape the endosome together and hear our plans to deliver therapeutic compounds to cardiomyocytes and modify the expression of genes causing cardiomyopathies.
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